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Lucia E. Rameh

Boston Biomedical Research Institute
Beth Israel Deaconess Medical Center

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Exposure of pancreatic ß-cells to excess glucose results in bimodal activation of mTORC1 and mTOR-dependent metabolic acceleration

2020 iScience
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Camptothecin resistance is determined by the regulation of topoisomerase I degradation mediated by ubiquitin proteasome pathway

2017 Oncotarget
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Analysis of the Phosphoinositide Composition of Subcellular Membrane Fractions

2016 Methods of Molecular Biology
- Deborah A. Sarkes
- Lucia E. Rameh
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IQGAP1 makes PI(3)K signalling as easy as PIP, PIP2, PIP3

2016 Nature Cell Biology
- Lucia E. Rameh
- Ashley M. Mackey
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Phosphoinositide signalling in type 2 diabetes: a β-cell perspective

2016 Biochemical Society Transactions
- Lucia E. Rameh
- Jude T. Deeney
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PIPPing on AKT1: How Many Phosphatases Does It Take to Turn off PI3K?

2015 Cancer Cell
- Alex Toker
- Lucia E. Rameh
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Abstract NG05: Depletion of a putatively druggable class of phosphatidylinositol kinases inhibits growth of p53 null tumors

2014 Cancer Research
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PIP4kγ is a substrate for mTORC1 that maintains basal mTORC1 signaling during starvation.

2014 Science Signaling
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Depletion of a Putatively Druggable Class of Phosphatidylinositol Kinases Inhibits Growth of p53-Null Tumors

2013 Cell
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AKT Facilitates EGFR Trafficking and Degradation by Phosphorylating and Activating PIKfyve

2013 Science Signaling
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