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Nerea Moreno

University of Valencia

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Preclinical characterization of antagomiR-218 as a potential treatment for Myotonic Dystrophy

2021 Molecular therapy. Nucleic acids
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Musashi-2 contributes to myotonic dystrophy muscle dysfunction by promoting excessive autophagy through miR-7 biogenesis repression.

2021 Molecular therapy. Nucleic acids
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miR-23b and miR-218 silencing increase Muscleblind-like expression and alleviate myotonic dystrophy phenotypes in mammalian models

2018 Nature Communications
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