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Woo-Sung Choi

University of British Columbia

6

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226

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A specific N-terminal residue in Kv1.5 is required for upregulation of the channel by SAP97.

2006 Biochemical and Biophysical Research Communications
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Effects of incretin hormones on β‐cell multiple delayed rectifier K+ channels

2006 The FASEB Journal
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Kv1.5 Surface Expression Is Modulated by Retrograde Trafficking of Newly Endocytosed Channels by the Dynein Motor

2005 Circulation Research
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A Novel Mechanism for the Suppression of a Voltage-gated Potassium Channel by Glucose-dependent Insulinotropic Polypeptide PROTEIN KINASE A-DEPENDENT ENDOCYTOSIS

2005 Journal of Biological Chemistry
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SAP97 increases Kv1.5 currents through an indirect N-terminal mechanism

2003 FEBS Letters
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Citations (53)