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Young Jin Gi

University of Texas MD Anderson Cancer Center

22

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Nucleus-mitochondria positive feedback loop formed by ERK5 S496 phosphorylation-mediated poly (ADP-ribose) polymerase activation provokes persistent pro-inflammatory senescent phenotype and accelerates coronary atherosclerosis after chemo-radiation.

2021 Redox biology
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Senescent Phenotype Induced by p90RSK-NRF2 Signaling Sensitizes Monocytes and Macrophages to Oxidative Stress in HIV+ Individuals: Implications for Atherogenesis

2019 Circulation
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Senescent Phenotype Induced by p90RSK-NRF2 Signaling Sensitizes Monocytes and Macrophages to Oxidative Stress in HIV-Positive Individuals

2019 Circulation
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Cancer Treatment (CTCT) Promotes Oxidative Stress and Alters Monocyte/ macrophage Biology

2019 Free Radical Biology and Medicine
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Senescent Phenotype Induced by p90RSK-NRF2 Signaling Sensitizes Monocytes and Macrophages to Oxidative Stress in HIV-Positive Individuals: Implications for Atherogenesis

2019 Circulation
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Endothelial senescence is induced by phosphorylation and nuclear export of telomeric repeat binding factor 2–interacting protein

2019 JCI insight
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Abstract 320: The Key Role of Endothelial Sumoylation in Regulating Sustained Nadph Oxidase Activation Under Disturbed Flow: Involvement of Feedback Loop Formed by Fak-sumoylation

2019 Arteriosclerosis, Thrombosis, and Vascular Biology
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Abstract 451: Radiation Induced Atherosclerosis on Mice

2018 Arteriosclerosis, Thrombosis, and Vascular Biology
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Abstract 223: Monocytes/macrophages Are Sensitized to Secondary Oxidative Insult by Cardiovascular Toxic Drugs via the P90rsk-erk5 S496 Phosphorylation Pathway, Aggrandizing Atherosclerotic Plaque Formation

2018 Arteriosclerosis, Thrombosis, and Vascular Biology
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Developing a Reliable Mouse Model for Cancer Therapy-Induced Cardiovascular Toxicity in Cancer Patients and Survivors

2018 Frontiers in Cardiovascular Medicine
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