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Brigitte Kasper

Hannover Medical School

14

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365

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Transfer of PBMC From SSc Patients Induces Autoantibodies and Systemic Inflammation in Rag2-/-/IL2rg-/- Mice.

2021 Frontiers in Immunology
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Expression of ACE2, TMPRSS2, and CTSL in human airway epithelial cells under physiological and pathological conditions: Implications for SARS-CoV2 infection

2020 bioRxiv
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Differential expression and regulation of GTPases (RhoA and Rac2) and GDIs (LyGDI and RhoGDI) in neutrophils from patients with severe congenital neutropenia

2000 Blood
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Association of src-kinase Lyn and non-src-kinase Syk with the granulocyte colony-stimulating factor receptor (G-CSFR) is not abrogated in neutrophils from severe congenital neutropenia patients with point mutations in the G-CSFR mRNA

1999 International Journal of Hematology
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Clinical implications of G-CSF receptor mutations

1998 Critical Reviews in Oncology Hematology
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Clinical Relevance of Point Mutations in the Cytoplasmic Domain of the Granulocyte Colony-Stimulating Factor Receptor Gene in Patients With Severe Congenital Neutropenia

1997 Blood
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Severe Congenital Neutropenia Patients With Point Mutations in the Granulocyte Colony-Stimulating Factor (G-CSF ) Receptor mRNA Express a Normal G-CSF Receptor Protein

1997 Blood
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Frequency of point mutations in the gene for the G-CSF receptor in patients with chronic neutropenia undergoing G-CSF therapy.

1997 Stem Cells
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The Protein Tyrosine Kinase JAK2 Is Activated In Neutrophils From Patients With Severe Congenital Neutropenia

1995 Blood
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Citations (36)