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Coral Ho

University of California, San Francisco

27

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1897

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Direct Comparison of SARS-CoV-2 Analytical Limits of Detection across Seven Molecular Assays.

2020 Journal of Clinical Microbiology
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Die Ko-Aktivierung von AKT and N-Ras in der Mäuseleber führt zu einer raschen Hepatokarzinogenese, vermittelt über mTORC1, FOXM1/SKP2 und c-Myc

2012 Zeitschrift Fur Gastroenterologie
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AKT (v-akt murine thymoma viral oncogene homolog 1) and N-Ras (neuroblastoma ras viral oncogene homolog) coactivation in the mouse liver promotes rapid carcinogenesis by way of mTOR (mammalian target of rapamycin complex 1), FOXM1 (forkhead box M1)/SKP2, and c-Myc pathways.

2012 Hepatology
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Bmi1 is required for hepatic progenitor cell expansion and liver tumor development.

2012 PLOS ONE
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Inactivation of Spry2 accelerates AKT-driven hepatocarcinogenesis via activation of MAPK and PKM2 pathways

2012 Journal of Hepatology
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Activation of the SKP2 protooncogene is a common effector pathway in hepatocarcinogenesis and contributes to malignant transformation of the liver

2011 Zeitschrift Fur Gastroenterologie
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Indian Hedgehog Regulates Intestinal Stem Cell Fate Through Epithelial−Mesenchymal Interactions During Development

2010 Gastroenterology
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Role of cyclin D1 as a mediator of c-Met- and beta-catenin-induced hepatocarcinogenesis.

2009 Cancer Research
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Chromosomal abnormalities determined by comparative genomic hybridization are helpful in the diagnosis of atypical hepatocellular neoplasms

2009 Histopathology
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Bmi1 Functions as an Oncogene Independent of Ink4A/Arf Repression in Hepatic Carcinogenesis

2009 Molecular Cancer Research
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Citations (57)