[Hepatorenal insufficiency syndrome in endotoxic shock (a light optical and electron microscopic study)].
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Experimental endotoxemia in the liver and kidneys was characterized by initial hemodynamic and rheologic disorders as well as intravascular coagulation disturbances some hours later. Hypoperfusion of these organs resulted in dystrophic changes in hepatocytes. Progression of endotoxin shock was accompanied by irreversible cellular changes. Late endotoxemia was characterized by adaptive changes associated with the improvement of microcirculation.Keywords:
Endotoxic shock
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Experimental endotoxemia in the liver and kidneys was characterized by initial hemodynamic and rheologic disorders as well as intravascular coagulation disturbances some hours later. Hypoperfusion of these organs resulted in dystrophic changes in hepatocytes. Progression of endotoxin shock was accompanied by irreversible cellular changes. Late endotoxemia was characterized by adaptive changes associated with the improvement of microcirculation.
Endotoxic shock
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Objective To discuss the relationship between myocardial microcirculation and myocardial perfusion after acute myocardial infarction.Methods Systematically analyze myocardial microcirculation pathophysiology,myocardial microcirculation in acute myocardial infarction,assessment methods of myocardial perfusion and therapy for improving myocardial perfusion.Results Local inflammatory stress and abnormal reaction of neurohumor lead myocardial microcirculation dysfunction,even complete occlusion,which damage myocardial perfusion.Conclusion The condition of myocardial microcirculation directly determined the level of myocardial perfusion after acute myocardial infarction.Worse myocardial perfusion predicted that re-myocardial infarction,congestive heart failure, malignant arrhythmia and cardiogenic sudden death increased significantly.
Coronary circulation
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showed cardiomegaly on chest X-ray.NYHA functional class>2 was found in 42.2% of TGA pts (NYHA class 3 in 9.3%) and in 50.0% of ccTGA pts (NYHA class 3 in 10.0%).Full criteria for CRT were met in 3.1% of the TGA pts and 5.0% of the ccTGA pts.Including pts in NYHA class 2 and without considering ventricular dilatation (ie.criteria similar to CONTAK CD) 6.3% of TGA and 5.0% of ccTGA pts would have been eligible for CRT.Conclusion: in unselected pts with a systemic right ventricle, 3.1% to 6.3% are potential candidates for CRT.If antibradycardia pacing is indicated, electrical resynchronization may also need to be considered.Prospective studies assessing the logistical and anatomical feasibility of CRT in ACHD cohorts with failing ventricles are warranted.
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Abnormality
Ventricular hypertrophy
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Cremaster muscle
Intravital microscopy
Blood cell
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This book is designed to provide a comprehensive picture of the areas of shock, ischemia, and anoxia, specifically covering new developments since 1965. It is divided into four sections, with 76 contributing authors. Section 1 deals with basic pathophysiological alterations at the level of the cell and in the microcirculation. An additional chapter on disseminated intravascular coagulation is included. Alterations in transport of calcium and other ions are emphasized. The second section is devoted specifically to shock, primarily at the organ level, and to its current therapy. Endotoxic, hemorrhagic, and septic shock are covered along with shock models, the use of computer registries, and consideration of such therapeutic modes as fluorocarbons and hyperbaric oxygen. The lung as a shock organ is extensively covered. Unfortunately, similar coverage is not given to the heart. Section 3 deals with injury to the CNS, both brain and spinal cord. While this topic seems to
Pathophysiology
Endotoxic shock
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Atrial natriuretic peptide
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Pathognomonic
Endotoxic shock
Backflow
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Background Endotoxic shock is one of clinical devastating symptom in common,which lead to the protopathy more serious and complexed.It has been paid more attention in the field of medicine because of its feral,rapid progress and high mortality.Objective This review summarized the therapeutic effect of electro-acupuncture in endotoxic shock.Content Electro-acupuncture plays an important role in stabling microcirculation,improving blood pressure,adjusting humoral factors and protecting important organs in endotoxic shock by inhibiting tumor necrosis factor (TNF)-α,interleukin (IL)-6,nitric oxide(NO) and oxygen free radical and so on.Trend The pathogenesis of endotoxic shock is complicated,and electro-acupuncture may be applied to remedy endotoxic shock and the damage of important organs by mobilizing endogenous protective system and the related signal transduction pathways.
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Electro-acupuncture; Endotoxic shock; Therapy
Endotoxic shock
Pathogenesis
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Contractility
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