DPP (Dipeptidyl Peptidase)-4 Inhibitor Attenuates Ang II (Angiotensin II)-Induced Cardiac Hypertrophy via GLP (Glucagon-Like Peptide)-1-Dependent Suppression of Nox (Nicotinamide Adenine Dinucleotide Phosphate Oxidase) 4-HDAC (Histone Deacetylase) 4 Pathway.

Kosuke Okabe,Shouji Matsushima,Soichiro Ikeda,Masataka Ikeda,Akihito Ishikita,Tomonori Tadokoro,Nobuyuki Enzan,Taishi Yamamoto,Masashi Sada,Hiroko Deguchi,Keisuke Shinohara,Tomomi Ide,Hiroyuki Tsutsui

DPP (Dipeptidyl Peptidase)-4 Inhibitor Attenuates Ang II (Angiotensin II)-Induced Cardiac Hypertrophy via GLP (Glucagon-Like Peptide)-1-Dependent Suppression of Nox (Nicotinamide Adenine Dinucleotide Phosphate Oxidase) 4-HDAC (Histone Deacetylase) 4 Pathway.

2020

Kosuke Okabe
Shouji Matsushima
Soichiro Ikeda
Masataka Ikeda
Akihito Ishikita
Tomonori Tadokoro
Nobuyuki Enzan
Taishi Yamamoto
Masashi Sada
Hiroko Deguchi
Keisuke Shinohara
Tomomi Ide
Hiroyuki Tsutsui

Nox4 (NADPH [Nicotinamide adenine dinucleotide phosphate] oxidase 4) is a major source of oxidative stress and is intimately involved in cardiac hypertrophy. DPP (Dipeptidyl peptidase)-4 inhibitor ...

Keywords:

NOX4
HDAC4
Nicotinamide adenine dinucleotide phosphate
Oxidase test
Medicine
Renin–angiotensin system
Dipeptidyl peptidase
Dipeptidyl peptidase-4 inhibitor
Endocrinology
Internal medicine
Angiotensin II

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