Endothelial Cell Dysfunction—Can One Outsmart Oxidative Stress by Direct Interaction with the Pathological Oxidized or Heme-Free Soluble Guanyl-Cyclase? Targeting the Heme-Oxydized Nitric Oxide Receptor for Selective Vasodilatation of Diseased Blood Vessels. J Clin Invest 116: 2552–2561, 2006

Johannes-Peter Stasch,Peter Schmidt,Pavel I. Nedvetsky,T.Y. Arun Nedvetskaya,H. S. Kumar,Sabine Meurer,Martin Deile,A Taye,Andreas Knorr,Harald Lapp,Helmut Müller,Y. Turgay,Christiane Rothkegel,Adrian Tersteegen,Barbara Kemp-Harper,Werner Müller–Esterl,Harald H.H.W. Schmidt

Endothelial Cell Dysfunction—Can One Outsmart Oxidative Stress by Direct Interaction with the Pathological Oxidized or Heme-Free Soluble Guanyl-Cyclase? Targeting the Heme-Oxydized Nitric Oxide Receptor for Selective Vasodilatation of Diseased Blood Vessels. J Clin Invest 116: 2552–2561, 2006

2007

Johannes-Peter Stasch
Peter Schmidt
Pavel I. Nedvetsky
T.Y. Arun Nedvetskaya
H. S. Kumar
Sabine Meurer
Martin Deile
A Taye
Andreas Knorr
Harald Lapp
Helmut Müller
Y. Turgay
Christiane Rothkegel
Adrian Tersteegen
Barbara Kemp-Harper
Werner Müller–Esterl
Harald H.H.W. Schmidt

![Figure][1] Eberhard Ritz Feature Editor It is well known that the rate of CV death is excessive in renal patients ([1][2]), but the underlying causes have not been completely resolved. Endothelial dysfunction, however, is thought to play a central role. One indicator of endothelial

Keywords:

Endothelial stem cell
Blood vessel
Heme
Endothelial dysfunction
Oxidative stress
Nitric oxide
Internal medicine
Endocrinology
Vasodilation
Medicine
Receptor
Diabetes mellitus

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