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Anja Krabbenhöft

Hannover Medical School

20

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Kir4.1 channel expression is essential for parietal cell control of acid secretion

2011 Journal of Biological Chemistry
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The switch of intestinal Slc26 exchangers from anion absorptive to HCO3- secretory mode is dependent on CFTR anion channel function

2010 American Journal of Physiology-cell Physiology
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mode is dependent on CFTR anion channel function 2

2010
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W1666 Genetic Deletion of NHE3, SLC26A6 (PAT1), SLC26A3 (DRA) and CFTR Helps Unravel the Different Pathways for Duodenal HCO3- and H+ Secretion In Vivo

2009 Gastroenterology
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1061 Gastric KNCQ1 Channels Provide Luminal K+ to the H+/K+ ATPase, Whereas Kir 4.1 Channels Are Involved in Secretory Membrane Recycling

2009 Gastroenterology
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Differential roles of NHERF1, NHERF2, and PDZK1 in regulating CFTR-mediated intestinal anion secretion in mice

2009 Journal of Clinical Investigation
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788 Effect of Genetic Ablation of the Acid/Base Transporters PNBC1, NHE1, NHE2, and Slc26a7, On Gastric Epithelial Functions

2009 Gastroenterology
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786 NBCn1 (Slc4a7) Is a Major HCO3- Importer in Murine Duodenum and Essential for Basal and Stimulated HCO3- Secretion In Vivo

2009 Gastroenterology
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Slc26 anion transporters involvement in acid-activated duodenal HCO 3 - secretion

2009
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NBCn1 (Slc4a7) is a major HCO3- importer in murine duodenum and essential for basal and stimulated HCO3- secretion in vivo

2009 The FASEB Journal
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