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Jana Hof

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19

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Identification of a genetically defined ultra-high-risk group in relapsed pediatric T-lymphoblastic leukemia

2017 Blood Cancer Journal
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Identification of an Ultra High-Risk and Targetable Molecular Signature in Relapsed Pediatric T-ALL

2016 Blood
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Pediatric T-cell lymphoblastic leukemia evolves into relapse by clonal selection, acquisition of mutations and promoter hypomethylation

2015 Haematologica
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Gene Panel Sequencing of Primary and Relapsed Pediatric T-ALL Shows That Relapse-Specific Mutations Are Diverse and Mostly Non-Recurrent

2015 Blood
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Ras pathway mutations are prevalent in relapsed childhood acute lymphoblastic leukemia and confer sensitivity to MEK inhibition. Blood . 2014;124(23):3420-3430

2015 Blood
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The activating STAT5B N642H mutation is a common abnormality in pediatric T-cell acute lymphoblastic leukemia and confers a higher risk of relapse

2014 Haematologica
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Clinical Significance of NT5C2 Mutations in Children with First Relapse of B-Cell Precursor Acute Lymphoblastic Leukemia

2014 Blood
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Activating mutations in the NT5C2 nucleotidase gene drive chemotherapy resistance in relapsed ALL

2013 Nature Medicine
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Mutations and Deletions of the TP53 Gene Predict Nonresponse to Treatment and Poor Outcome in First Relapse of Childhood Acute Lymphoblastic Leukemia

2011 Journal of Clinical Oncology
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High VLA-4 expression is associated with adverse outcome and distinct gene expression changes in childhood B-cell precursor acute lymphoblastic leukemia at first relapse

2011 Haematologica
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