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Lindsey M. Hutt-Fletcher

LSU Health Sciences Center Shreveport
University of Missouri–Kansas City

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The BDLF3 gene product of Epstein-Barr virus, gp150, mediates non-productive binding to heparan sulfate on epithelial cells and only the binding domain of CD21 is required for infection.

2016 Virology
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The Epstein-Barr Virus Glycoprotein gp150 Forms an Immune-Evasive Glycan Shield at the Surface of Infected Cells.

2016 PLOS Pathogens
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Nonmuscle myosin heavy chain IIA mediates Epstein-Barr virus infection of nasopharyngeal epithelial cells.

2015 Proceedings of the National Academy of Sciences of the United States of America
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Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells

2015 PLOS Pathogens
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Neuropilin 1 is an entry factor that promotes EBV infection of nasopharyngeal epithelial cells

2015 Nature Communications
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Epstein–Barr virus replicating in epithelial cells

2014 Proceedings of the National Academy of Sciences of the United States of America
- Lindsey M. Hutt-Fletcher
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Epstein-Barr Virus Glycoprotein gB and gHgL Can Mediate Fusion and Entry in trans, and Heat Can Act as a Partial Surrogate for gHgL and Trigger a Conformational Change in gB

2014 Journal of Virology
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αvβ6- and αvβ8-Integrins Serve As Interchangeable Receptors for HSV gH/gL to Promote Endocytosis and Activation of Membrane Fusion

2013 PLOS Pathogens
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Lymphoma Apoptosis, and Tumorigenicity in Burkitt Epstein-Barr Virus Regulates c-MYC,

2013
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Oral dysplasia and squamous cell carcinoma: Correlation between increased expression of CD21, Epstein-Barr virus and CK19

2012 Oral Oncology
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Citations (27)