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Yutaka Sato

Nathan Kline Institute for Psychiatric Research

10

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980

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Lysosomal dysfunction in Down syndrome is APP-dependent and mediated by APP-βCTF (C99)

2019 The Journal of Neuroscience
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mTOR hyperactivation in Down Syndrome underlies deficits in autophagy induction, autophagosome formation, and mitophagy

2019 Cell Death and Disease
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Evidence that the rab5 effector APPL1 mediates APP-βCTF-induced dysfunction of endosomes in Down syndrome and Alzheimer's disease.

2016 Molecular Psychiatry
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Partial BACE1 reduction in a Down syndrome mouse model blocks Alzheimer-related endosomal anomalies and cholinergic neurodegeneration: role of APP-CTF

2016 Neurobiology of Aging
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Neurofilament subunits are integral components of synapses and modulate neurotransmission and behavior in vivo.

2015 Molecular Psychiatry
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Functions of neurofilaments in synapses

2015 Molecular Psychiatry
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Presenilin 1 Maintains Lysosomal Ca2+ Homeostasis via TRPML1 by Regulating vATPase-Mediated Lysosome Acidification

2015 Cell Reports
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Single-Walled Carbon Nanotubes Alleviate Autophagic/Lysosomal Defects in Primary Glia from a Mouse Model of Alzheimer’s Disease

2014 Nano Letters
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Lysosomal Proteolysis Inhibition Selectively Disrupts Axonal Transport of Degradative Organelles and Causes an Alzheimer's-Like Axonal Dystrophy

2011 The Journal of Neuroscience
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Primary lysosomal dysfunction causes cargo-specific deficits of axonal transport leading to Alzheimer-like neuritic dystrophy.

2011 Autophagy
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Citations (63)