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Chiaki Omura

Northwestern University

14

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1983

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Pharmacologic rescue of circadian β-cell failure through P2Y1 purinergic receptor identified by small-molecule screen

2021 bioRxiv
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A role for alternative splicing in circadian control of exocytosis and glucose homeostasis

2020 Genes & Development
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Transcriptional Basis for Rhythmic Control of Hunger and Metabolism within the AgRP Neuron

2019 Cell Metabolism
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Requirement for NF-κB in maintenance of molecular and behavioral circadian rhythms in mice

2018 Genes & Development
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Author response: Usf1, a suppressor of the circadian Clock mutant, reveals the nature of the DNA-binding of the CLOCK:BMAL1 complex in mice

2013 eLife
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Shedding new light on circadian clocks Using a clever combination of genetic and biochemical tools researchers have shown that a transcription factor called USF1 has a central role in determining how mutations of the Clock gene manifest themselves in the behaviour of different mouse strains.

2013
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Disruption of the clock components CLOCK and BMAL1 leads to hypoinsulinaemia and diabetes

2010 Nature
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Genetic suppression of the circadian Clock mutation by the melatonin biosynthesis pathway

2010 Proceedings of the National Academy of Sciences of the United States of America
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Generation, identification and functional characterization of the nob4 mutation of Grm6 in the mouse

2007 Visual Neuroscience
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