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Mikiko Iguchi-Hashimoto
Mikiko Iguchi-Hashimoto
Kyoto University
Medicine
Biology
Molecular biology
Interleukin 17
Severity of illness
4
Papers
213
Citations
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The association between serious infection and disease outcome in patients with rheumatoid arthritis
2016
Clinical Rheumatology
Mikiko Iguchi-Hashimoto
Motomu Hashimoto
Takao Fujii
Masahide Hamaguchi
Moritoshi Furu
Masahiro Ishikawa
Hiromu Ito
Noriyuki Yamakawa
Chikashi Terao
Keiichi Yamamoto
Wataru Yamamoto
Koichiro Ohmura
Tsuneyo Mimori
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Citations (9)
Follistatin‐related protein/follistatin‐like 1 evokes an innate immune response via CD14 and toll‐like receptor 4
2012
FEBS Letters
Kosaku Murakami
Masao Tanaka
Takashi Usui
Daisuke Kawabata
Aoi Shiomi
Mikiko Iguchi-Hashimoto
Masakazu Shimizu
Naoichiro Yukawa
Hajime Yoshifuji
Takaki Nojima
Koichiro Ohmura
Takao Fujii
Hisanori Umehara
Tsuneyo Mimori
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Citations (46)
Overexpression of a Minimal Domain of Calpastatin Suppresses IL-6 Production and Th17 Development via Reduced NF-κB and Increased STAT5 Signals
2011
PLOS ONE
Mikiko Iguchi-Hashimoto
Takashi Usui
Hajime Yoshifuji
Masakazu Shimizu
Shio Kobayashi
Yoshinaga Ito
Kosaku Murakami
Aoi Shiomi
Naoichiro Yukawa
Daisuke Kawabata
Takaki Nojima
Koichiro Ohmura
Takao Fujii
Tsuneyo Mimori
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Citations (18)
Gamma/delta T cells are the predominant source of interleukin‐17 in affected joints in collagen‐induced arthritis, but not in rheumatoid arthritis
2009
Arthritis & Rheumatism
Yoshinaga Ito
Takashi Usui
Shio Kobayashi
Mikiko Iguchi-Hashimoto
Hiromu Ito
Hiroyuki Yoshitomi
Takashi Nakamura
Masakazu Shimizu
Daisuke Kawabata
Naoichiro Yukawa
Motomu Hashimoto
Noriko Sakaguchi
Shimon Sakaguchi
Hajime Yoshifuji
Takaki Nojima
Koichiro Ohmura
Takao Fujii
Tsuneyo Mimori
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Citations (140)
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