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Mindong Ren

New York University

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Cardiolipin remodeling enables protein crowding in the inner mitochondrial membrane.

2021 The EMBO Journal
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Neurological & psychological aspects of Barth syndrome: Clinical manifestations and potential pathogenic mechanisms.

2021 Mitochondrion
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A simple mechanistic explanation for Barth syndrome and cardiolipin remodeling.

2021 Journal of Inherited Metabolic Disease
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Substantial Decrease in Plasmalogen in the Heart Associated with Tafazzin Deficiency

2018 Biochemistry
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The Basis for Acyl Specificity in the Tafazzin Reaction

2017 Journal of Biological Chemistry
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The PPAR pan-agonist bezafibrate ameliorates cardiomyopathy in a mouse model of Barth syndrome

2017 Orphanet Journal of Rare Diseases
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Loss of protein association causes cardiolipin degradation in Barth syndrome

2016 Nature Chemical Biology
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Content of Plasmalogen Lipids Markedly Decreases in Barth Syndrome

2016 Biophysical Journal
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cardiovascular development Ultrasound biomicroscopy-Doppler in mouse

2015
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Tafazzins from Drosophila and mammalian cells assemble in large protein complexes with a short half-life.

2015 Mitochondrion
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