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F ORourke

University of Connecticut Health Center

9

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389

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Cloning of human Ca2+ homoeostasis endoplasmic reticulum protein (CHERP): regulated expression of antisense cDNA depletes CHERP, inhibits intracellular Ca2+ mobilization and decreases cell proliferation.

2000 Biochemical Journal
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Immunohistochemical localization of the InsP4 receptor GTPase-activating protein GAP1IP4BP in the rat brain †

1999 Journal of Neuroscience Research
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Antisense Knock Out of the Inositol 1,3,4,5-Tetrakisphosphate Receptor GAP1IP4BP in the Human Erythroleukemia Cell Line Leads to the Appearance of Intermediate Conductance K(Ca) Channels that Hyperpolarize the Membrane and Enhance Calcium Influx

1999 The Journal of General Physiology
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The inositol 1,4,5-trisphosphate receptor binding sites of platelet membranes. pH-dependency, inhibition by polymeric sulphates, and the possible presence of arginine at the binding site.

1990 Biochemical Journal
- F ORourke
- Maurice B. Feinstein
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Release of Ca2+ by inositol 1,4,5-trisphosphate in platelet membrane vesicles is not dependent on cyclic AMP-dependent protein kinase

1989 Biochemical Journal
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Stimulus-response coupling in a cell-free platelet membrane system: GTP-dependent release of Ca2+ by thrombin, and inhibition by pertussis toxin and a monoclonal antibody that blocks calcium release by IP3

1987 FEBS Letters
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Inositol 1,4,5-trisphosphate releases Ca2+ from a Ca2+-transporting membrane vesicle fraction derived from human platelets.

1985 Journal of Biological Chemistry
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Citations (232)