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James R. Groome

Idaho State University

28

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311

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Charge substitutions at the voltage-sensing module of domain III enhance actions of site-3 and site-4 toxins on an insect sodium channel.

2021 Insect Biochemistry and Molecular Biology
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Markov and Eyring Modeling Predict Differential Contributions of Homologous Mutations in Hypokalemic Periodic Paralysis to Skeletal Muscle Fiber Excitability

2021 Biophysical Journal
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Cysteine Mutagenesis to Probe Site-Specific Interactions in the Voltage Sensor Module of hNaV1.4

2020 Biophysical Journal
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Ribbon α-Conotoxin KTM Exhibits Potent Inhibition of Nicotinic Acetylcholine Receptors

2019 Marine Drugs
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Negative Countercharges and S4 Interaction in Domain IV of Nav1.4

2014 Biophysical Journal
- James R. Groome
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Domain III S4 in closed-state fast inactivation: Insights from a periodic paralysis mutation

2014 Channels
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NaV1.4 mutations cause hypokalaemic periodic paralysis by disrupting IIIS4 movement during recovery

2014 Brain
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S1–S3 counter charges in the voltage sensor module of a mammalian sodium channel regulate fast inactivation

2013 The Journal of General Physiology
- James R. Groome
- Vern Winston
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Pathophysiological role of omega pore current in channelopathies

2012 Frontiers in Pharmacology
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Voltage Sensor Module in NaV1.4: S1-S3 Charges Regulate Fast Inactivation

2012 Biophysical Journal
- James R. Groome
- Vern Winston
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