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Virendra N. Pandey

Rutgers University

75

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Correction for Dixit et al., “FUSE Binding Protein 1 Facilitates Persistent Hepatitis C Virus Replication in Hepatoma Cells by Regulating Tumor Suppressor p53”

2017 Journal of Virology
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The β1′−β2′ Motif of the RNase H Domain of Human Immunodeficiency Virus Type 1 Reverse Transcriptase Is Responsible for Conferring Open Conformation to the p66 Subunit by Displacing the Connection Domain from the Polymerase Cleft

2017 Biochemistry
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Modulation of HCV replication and translation by ErbB3 binding protein1 isoforms

2017 Virology
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HCV-Host Cell Interactions: Role of Ebp-1 in Hepatitis C virus Replication

2015 The FASEB Journal
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FUSE Binding Protein 1 Facilitates Persistent Hepatitis C Virus Replication in Hepatoma Cells by Regulating Tumor Suppressor p53

2015 Journal of Virology
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Fuse binding protein antagonizes the transcription activity of tumor suppressor protein p53

2014 BMC Cancer
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Affinity Capture and Identification of Host Cell Factors Associated with Hepatitis C Virus (+) Strand Subgenomic RNA

2013 Molecular & Cellular Proteomics
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Affinity labeling of hepatitis C virus replicase with a nucleotide analogue: identification of binding site.

2013 Biochemistry
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Identification and evaluation of anti Hepatitis C Virus phytochemicals from Eclipta alba

2012 Journal of Ethnopharmacology
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The glutamine side chain at position 91 on the β5a-β5b loop of human immunodeficiency virus type 1 reverse transcriptase is required for stabilizing the dNTP binding pocket.

2011 Biochemistry
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