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Mark M. Yore

Dartmouth College

26

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Retinol binding protein 4 primes the NLRP3 inflammasome by signaling through Toll-like receptors 2 and 4.

2020 Proceedings of the National Academy of Sciences of the United States of America
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Methodological Issues in Studying PAHSA Biology: Masking PAHSA Effects

2018 Cell Metabolism
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CTLA4-Ig treatment improves RBP4-induced adipose tissue inflammation and insulin resistance triggered by MyD88, JNK, ERK and p38 pathways (IRC8P.443)

2015 Journal of Immunology
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Discovery of a class of endogenous mammalian lipids with anti-diabetic and anti-inflammatory effects.

2014 Cell
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RBP4 Activates Antigen-Presenting Cells, Leading to Adipose Tissue Inflammation and Systemic Insulin Resistance

2014 Cell Metabolism
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Retinol-Binding Protein 4 Inhibits Insulin Signaling in Adipocytes by Inducing Proinflammatory Cytokines in Macrophages through a c-Jun N-Terminal Kinase- and Toll-Like Receptor 4-Dependent and Retinol-Independent Mechanism

2012 Molecular and Cellular Biology
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New synthetic triterpenoids: potent agents for prevention and treatment of tissue injury caused by inflammatory and oxidative stress.

2011 Journal of Natural Products
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Proteomic Analysis Shows Synthetic Oleanane Triterpenoid Binds to mTOR

2011 PLOS ONE
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Triterpenoids CDDO-Methyl Ester or CDDO-Ethyl Amide and Rexinoids LG100268 or NRX194204 for Prevention and Treatment of Lung Cancer in Mice

2009 Cancer Prevention Research
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The rexinoid LG100268 and the synthetic triterpenoid CDDO-methyl amide are more potent than erlotinib for prevention of mouse lung carcinogenesis

2008 Molecular Cancer Therapeutics
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