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Jianhua Zhang

University of Texas Health Science Center at San Antonio

31

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808

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Aliskiren and Valsartan Mediate Left Ventricular Remodeling Post-Myocardial Infarction in Mice through MMP-9 Effects

2014 Journal of Molecular and Cellular Cardiology
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Obese and diabetic KKAy mice show increased mortality but improved cardiac function following myocardial infarction

2013 Cardiovascular Pathology
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Cardiac Wound Healing Post-myocardial Infarction: A Novel Method to Target Extracellular Matrix Remodeling in the Left Ventricle

2013 Methods of Molecular Biology
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Matrix Metalloproteinase-28 Deletion Exacerbates Cardiac Dysfunction and Rupture After Myocardial Infarction in Mice by Inhibiting M2 Macrophage Activation

2013 Circulation Research
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MMP-9 overexpression in macrophages regulates the post-myocardial infarction inflammatory response through SCYE1

2012 The FASEB Journal
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Chronic and intermittent hypoxia differentially regulate left ventricular inflammatory and extracellular matrix responses

2012 Hypertension Research
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Matrix Metalloproteinase-28 Deletion Exacerbates Cardiac Dysfunction and Rupture Following Myocardial Infarction in Mice by Inhibiting M2 Macrophage Activation

2012 Circulation Research
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Matrix metalloproteinase-9 deletion attenuates myocardial fibrosis and diastolic dysfunction in ageing mice

2012 Cardiovascular Research
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Transgenic overexpression of matrix metalloproteinase-9 in macrophages attenuates the inflammatory response and improves left ventricular function post-myocardial infarction.

2012 Journal of Molecular and Cellular Cardiology
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Matrix Metalloproteinase‐28 Deletion Attenuates Short‐term Left Ventricular Dysfunction but Exacerbates Cardiac Rupture Post‐Myocardial Infarction in Mice

2012 The FASEB Journal
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