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Trisha S. Smith

University of Virginia

9

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1066

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Pramipexole reduces reactive oxygen species production in vivo and in vitro and inhibits the mitochondrial permeability transition produced by the parkinsonian neurotoxin methylpyridinium ion.

2002 Journal of Neurochemistry
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Neurotoxic Aβ peptides increase oxidative stress in vivo through NMDA-receptor and nitric-oxide-synthase mechanisms, and inhibit complex IV activity and induce a mitochondrial permeability transition in vitro

2001 Journal of Neurochemistry
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Effects of single and multiple treatments with L-dihydroxyphenylalanine (L-DOPA) on dopamine receptor-G protein interactions and supersensitive immediate early gene responses in striata of rats after reserpine treatment or with unilateral nigrostriatal lesions.

1999 Journal of Neuroscience Research
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Elevated reactive oxygen species and antioxidant enzyme activities in animal and cellular models of Parkinson's disease

1997 Biochimica et Biophysica Acta
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Mitochondrial toxins in models of neurodegenerative diseases. II: elevated zif268 transcription and independent temporal regulation of striatal D1 and D2 receptor mRNAs and D1 and D2 receptor-binding sites in C57BL/6 mice during MPTP treatment

1997 Brain Research
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Mitochondrial toxins in models of neurodegenerative diseases. I: in vivo brain hydroxyl radical production during sytemic MPTP treatment or following microdialysis infusion of methylpyridinium or azide ions

1997 Brain Research
- Trisha S. Smith
- James P. Bennett
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Dopamine Neurons from Transgenic Mice with a Knockout of the p53 Gene Resist MPTP Neurotoxicity

1996 Neurodegeneration
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Reduction of Mpp+-induced hydroxyl radical formation and nigrostriatal Mptp toxicity by inhibiting nitric oxide synthase

1994 Neuroreport
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L-DOPA increases nigral production of hydroxyl radicals in vivo: potential L-DOPA toxicity?

1994 Neuroreport
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Citations (97)