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Kimberly A. Kelly

National Institute for Occupational Safety and Health

16

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Modeling Neuroimmune Interactions in Human Subjects and Animal Models to Predict Subtype-Specific Multidrug Treatments for Gulf War Illness.

2021 International Journal of Molecular Sciences
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The β-adrenergic receptor blocker and anti-inflammatory drug propranolol mitigates brain cytokine expression in a long-term model of Gulf War Illness.

2021 Life Sciences
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Acetylcholinesterase inhibitor exposures as an initiating factor in the development of Gulf War Illness, a chronic neuroimmune disorder in deployed veterans.

2020 Neuropharmacology
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Astrocyte-specific transcriptome analysis using the ALDH1L1 bacTRAP mouse reveals novel biomarkers of astrogliosis in response to neurotoxicity

2019 Journal of Neurochemistry
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Corticosterone potentiates DFP-induced neuroinflammation and affects high-order diffusion imaging in a rat model of Gulf War Illness

2018 Brain Behavior and Immunity
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Epigenetic impacts of stress priming of the neuroinflammatory response to sarin surrogate in mice: a model of Gulf War illness

2018 Journal of Neuroinflammation
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A Logic Model of Neuronal-Glial Interaction Suggests Altered Homeostatic Regulation in the Perpetuation of Neuroinflammation

2018 Frontiers in Cellular Neuroscience
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Corticosterone and exogenous glucose alter blood glucose levels, neurotoxicity, and vascular toxicity produced by methamphetamine

2017 Journal of Neurochemistry
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Corticosterone primes the neuroinflammatory response to Gulf War Illness‐relevant organophosphates independently of acetylcholinesterase inhibition

2017 Journal of Neurochemistry
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Chronic corticosterone primes the brain response to select neuroinflammatory agents by overexpression of toll-like receptor 2 and S100A8: A potential role for microglia

2015
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