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Gianluca Gitti

Wayne State University

10

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358

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C and smac/DIABLO XIAP to smac/DIABLO, and reduces the mitochondrial leakage of cytochrome Minocycline inhibits caspase activation and reactivation, increases the ratio of

2010
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apoptosis associated with increased urocortin expression in viable cells Warm blood cardioplegic arrest induces mitochondrial-mediated cardiomyocyte

2010
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Therapeutic modulation of the nitric oxide: all ace inhibitors are not equivalent

2007 Pharmacological Research
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Experimental Ischemic Cardiomyopathy: Insights into Remodeling, Physiological Adaptation, and Humoral Response

2006 Annals of Clinical and Laboratory Science
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Effect of nolomirole on monocrotaline-induced heart failure.

2004 Pharmacological Research
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Warm blood cardioplegic arrest induces mitochondrial-mediated cardiomyocyte apoptosis associated with increased urocortin expression in viable cells

2004 The Journal of Thoracic and Cardiovascular Surgery
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Minocycline inhibits caspase activation and reactivation, increases the ratio of XIAP to smac/DIABLO, and reduces the mitochondrial leakage of cytochrome C and smac/DIABLO

2004 Journal of the American College of Cardiology
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Epigallocatechin-3-gallate inhibits STAT-1 activation and protects cardiac myocytes from ischemia/reperfusion-induced apoptosis

2004 The FASEB Journal
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Thoracic and Cardiovascular

2003
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Minocycline, a novel cardioprotective agent, inhibits caspase activation and reactivation by increasing the ratio of XIAP to Smac/DIABLO and reducing mitochondrial leakage of cytochrome C and Smac/DIABLO

2003
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