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Jose R. Bayascas

Autonomous University of Barcelona

37

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Gossypol Treatment Restores Insufficient Apoptotic Function of DFF40/CAD in Human Glioblastoma Cells.

2021 Cancers
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Effects of CK2β subunit down-regulation on Akt signalling in HK-2 renal cells

2020 PLOS ONE
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Reducing the Levels of Akt Activation by PDK1 Knock-in Mutation Protects Neuronal Cultures against Synthetic Amyloid-Beta Peptides

2018 Frontiers in Aging Neuroscience
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Mutation of the 3-phosphoinositide-dependent protein kinase-1 (PDK1) substrate-docking site in the developing brain causes microcephaly with abnormal brain morphogenesis independently of Akt, leading to impaired cognition and disruptive behaviors.

2016 Molecular and Cellular Biology
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Correction for Zurashvili et al., Interaction of PDK1 with Phosphoinositides Is Essential for Neuronal Differentiation but Dispensable for Neuronal Survival

2016 Molecular and Cellular Biology
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The New Antitumor Drug ABTL0812 Inhibits the Akt/mTORC1 Axis by Upregulating Tribbles-3 Pseudokinase

2016 Clinical Cancer Research
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Fine-tuning the intensity of the PKB/Akt signal enables diverse physiological responses

2014 Cell Cycle
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Canonical and Kinase Activity-Independent Mechanisms for Extracellular Signal-Regulated Kinase 5 (ERK5) Nuclear Translocation Require Dissociation of Hsp90 from the ERK5-Cdc37 Complex

2013 Molecular and Cellular Biology
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Interaction of PDK1 with Phosphoinositides Is Essential for Neuronal Differentiation but Dispensable for Neuronal Survival

2013 Molecular and Cellular Biology
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S6K1 activation is not required for muscle hypertrophy

2011 The FASEB Journal
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