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Andrea D. Eckhart

Thomas Jefferson University

76

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Persistent increases in Ca 2+ influx through Cav1.2 shortens action potential and causes Ca 2+ overload-induced afterdepolarizations and arrhythmias

2016 Basic Research in Cardiology
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Untargeted metabolomic analysis for the clinical screening of inborn errors of metabolism

2015 Journal of Inherited Metabolic Disease
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Metabolomics as a Key Integrator for “Omic” Advancement of Personalized Medicine and Future Therapies

2012 Clinical and Translational Science
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S100A1 Genetically Targeted Therapy Reverses Dysfunction of Human Failing Cardiomyocytes

2011 Journal of the American College of Cardiology
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Calcium Influx through Cav1.2 Is a Proximal Signal for Pathological Cardiomyocyte Hypertrophy

2011 Journal of Molecular and Cellular Cardiology
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Impaired neoangiogenesis in β2-adrenoceptor gene-deficient mice: restoration by intravascular human β2-adrenoceptor gene transfer and role of NFκB and CREB transcription factors

2011 British Journal of Pharmacology
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The inotropic peptide ?ARKct improves ?AR responsiveness in normal and failing cardiomyocytes through G(??)-mediated L-type calcium current disinhibition

2011 Circulation Research
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The inotropic peptide βARKct improves βAR responsiveness in normal and failing cardiomyocytes through G(βγ)-mediated L-type calcium current disinhibition.

2011 Circulation Research
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Regulation of GPCR signaling in Hypertension

2010 Biochimica et Biophysica Acta
- Henriette Brinks
- Andrea D. Eckhart
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Level of G protein–Coupled Receptor Kinase-2 Determines Myocardial Ischemia/Reperfusion Injury via Pro- and Anti-Apoptotic Mechanisms

2010 Circulation Research
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