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David Kabat

St. Jude Children's Research Hospital

46

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Kinetic mechanism for HIV-1 neutralization by antibody 2G12 entails reversible glycan binding that slows cell entry

2012 Proceedings of the National Academy of Sciences of the United States of America
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Human immunodeficiency virus type 1 Vif functionally interacts with diverse APOBEC3 cytidine deaminases and moves with them between cytoplasmic sites of mRNA metabolism.

2008 Journal of Virology
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The Anti-HIV-1 Editing Enzyme APOBEC3G Binds HIV-1 RNA and Messenger RNAs That Shuttle between Polysomes and Stress Granules

2006 Journal of Biological Chemistry
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Regulated Production and Anti-HIV Type 1 Activities of Cytidine Deaminases APOBEC3B, 3F, and 3G

2005 AIDS Research and Human Retroviruses
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Kinetic Factors Control Efficiencies of Cell Entry, Efficacies of Entry Inhibitors, and Mechanisms of Adaptation of Human Immunodeficiency Virus

2005 Journal of Virology
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Variants of Human Immunodeficiency Virus Type 1 That Efficiently Use CCR5 Lacking the Tyrosine-Sulfated Amino Terminus Have Adaptive Mutations in gp120, Including Loss of a Functional N-Glycan

2005 Journal of Virology
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A process for the identification of inhibitors

2004
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Transcriptional regulation of APOBEC3G, a cytidine deaminase that hypermutates human immunodeficiency virus.

2004 Journal of Biological Chemistry
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Segregation of CD4 and CXCR4 into Distinct Lipid Microdomains in T Lymphocytes Suggests a Mechanism for Membrane Destabilization by Human Immunodeficiency Virus

2002 Journal of Virology
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Adaptive Mutations in the V3 Loop of gp120 Enhance Fusogenicity of Human Immunodeficiency Virus Type 1 and Enable Use of a CCR5 Coreceptor That Lacks the Amino-Terminal Sulfated Region

2001 Journal of Virology
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Citations (44)