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Ann-Christin Brorsson

Linköping University

37

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The Luminescent Conjugated Oligothiophene h-FTAA Attenuates the Toxicity of Different Aβ Species.

2021 Biochemistry
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Exploring Aβ Proteotoxicity and Therapeutic Candidates Using Drosophila melanogaster

2021 International Journal of Molecular Sciences
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Mapping pathogenic processes contributing to neurodegeneration in Drosophila models of Alzheimer’s disease

2020 FEBS Open Bio
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Serum amyloid P component promotes formation of distinct aggregated lysozyme morphologies and reduces toxicity in Drosophila flies expressing F57I lysozyme

2020 PLOS ONE
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MODELLING AD: THE APP-BACE1 FLY MODEL AND ITS POTENTIAL AS AN IN VIVO SYSTEM FOR STUDYING DISEASE-MODULATING COMPOUNDS

2017 Alzheimers & Dementia
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Serum Amyloid P Component Ameliorates Neurological Damage Caused by Expressing a Lysozyme Variant in the Central Nervous System of Drosophila melanogaster

2016 PLOS ONE
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Beneficial effects of increased lysozyme levels in Alzheimer’s disease modelled in Drosophila melanogaster

2016 FEBS Journal
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The luminescent oligothiophene p-FTAA converts toxic Aβ1-42 species into nontoxic amyloid fibers with altered properties

2016 Journal of Biological Chemistry
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Protective properties of lysozyme on β-amyloid pathology: implications for Alzheimer disease.

2015 Neurobiology of Disease
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Co-expression of a disease-associated lysozyme variant with human lysozyme in Drosophila causes accumulation of amyloid deposits and neurodegeneration

2014
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