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Fen-Biao Gao

University of Massachusetts Medical School
University of California, San Francisco

122

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19238

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Ribosome Profiling Reveals Novel Regulation of C9ORF72 GGGGCC Repeat-Containing RNA Translation.

2021 RNA
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A C. elegans model of C9orf72-associated ALS/FTD uncovers a conserved role for eIF2D in RAN translation

2021 Nature Communications
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Translation of the Poly(GR) Frame in C9ORF72-ALS/FTD Is Regulated by Cis-Elements Involved in Alternative Splicing

2021 Neurobiology of Aging
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TBK1 haploinsufficiency in ALS and FTD compromises membrane trafficking.

2021 Acta Neuropathologica
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p53 is a central regulator driving neurodegeneration caused by C9orf72 poly(PR).

2021 Cell
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Altered MICOS Morphology and Mitochondrial Ion Homeostasis Contribute to Poly(GR) Toxicity Associated with C9-ALS/FTD.

2020 Cell Reports
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Premature termination codon readthrough upregulates progranulin expression and improves lysosomal function in preclinical models of GRN deficiency.

2020 Molecular Neurodegeneration
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Activated iPSC-microglia from C9orf72 ALS/FTD patients exhibit endosomal-lysosomal dysfunction

2020 bioRxiv
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Ik2/TBK1 and Hook/Dynein, an adaptor complex for early endosome transport, are genetic modifiers of FTD-associated mutant CHMP2B toxicity in Drosophila.

2020 Scientific Reports
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CRISPR deletion of the C9ORF72 promoter in ALS/FTD patient motor neurons abolishes production of dipeptide repeat proteins and rescues neurodegeneration.

2020 Acta Neuropathologica
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