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Emanuel Berger

Technische Universität München

18

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539

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Mitochondrial impairment drives intestinal stem cell transition into dysfunctional Paneth cells predicting Crohn’s disease recurrence

2020 Gut
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120 – Mitochondrial Impairment in Crohn's Disease-Like Inflammation Drives Intestinal Stem Cell Transition Towards Dysfunctional Paneth Cells

2019 Gastroenterology
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Mitochondrial function controls intestinal epithelial stemness and proliferation

2016 Nature Communications
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403 Tissue-Specific Induction of Activating Transcription Factor 6 Signaling Promotes Intestinal Dysbiosis and Colonic Tumorigenesis

2015 Gastroenterology
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C/EBP homologous protein inhibits tissue repair in response to gut injury and is inversely regulated with chronic inflammation.

2014 Mucosal Immunology
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630 Expression of C/EBP Homologous Protein in Intestinal Epithelial Cells is Not Associated With the Development of Chronic Inflammation: A Transgenic Approach

2012 Gastroenterology
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Induction of dsRNA-activated protein kinase links mitochondrial unfolded protein response to the pathogenesis of intestinal inflammation

2012 Gut
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Endoplasmic Reticulum Stress Response Promotes Cytotoxic Phenotype of CD8αβ+ Intraepithelial Lymphocytes in a Mouse Model for Crohn’s Disease-like Ileitis

2012 Journal of Immunology
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M1826 Chemical Chaperones Modulate Endoplasmic Reticulum Stress Pathways in Intestinal Epithelial Cells

2010 Gastroenterology
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84 Mitochondrial Stress Induction in the Epithelium Fuels Endoplasmic Reticulum Unfolded Protein Responses via Double-Stranded RNA-Activated Protein Kinase (PKR) Under Conditions of Chronic Intestinal Inflammation

2010 Gastroenterology
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