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Sara Gastoldi

Mario Negri Institute for Pharmacological Research

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SARS-CoV-2 Spike Protein 1 Activates Microvascular Endothelial Cells and Complement System Leading to Thrombus Formation

2021 Social Science Research Network
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Molecular Studies and an ex vivo Complement Assay on Endothelium Highlight the Genetic Complexity of Atypical Hemolytic Uremic Syndrome: The Case of a Pedigree With a Null CD46 Variant

2020 Frontiers in Medicine
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Factor H Competitor Generated by Gene Conversion Events Associates with Atypical Hemolytic Uremic Syndrome

2017 Journal of The American Society of Nephrology
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Prevalent FHR-1 mutant protein generated by gene conversion reveals crucial role of factor H polymorphisms in atypical Hemolytic Uremic Syndrome (aHUS)

2016 Immunobiology
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A complicated case of atypical hemolytic uremic syndrome with frequent relapses under eculizumab.

2015 Pediatric Nephrology
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Dynamics of complement activation in aHUS and how to monitor eculizumab therapy

2014 Blood
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This information is current as Activation in Disease Models Proconvertase and Inhibits Complement that Blocks Formation of the C3bB A Novel Antibody against Human Factor B

2014
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ADAMTS13 Predicts Renal and Cardiovascular Events in Type 2 Diabetic Patients and Response to Therapy

2013 Diabetes
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C5a/C5aR interaction mediates complement activation and thrombosis on endothelial cells in atypical hemolytic uremic syndrome (aHUS)

2012 Immunobiology
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Alternative pathway activation of complement by Shiga toxin promotes exuberant C3a formation that triggers microvascular thrombosis.

2011 Journal of Immunology
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