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Uri Seligsohn

Sheba Medical Center
Tel Aviv University

261

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Platelets from Calreticulin Mutated Essential Thrombocythemia Patients are Less Reactive than JAK2 V617F Mutated Platelets

2020 American Journal of Hematology
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The reduced form of coagulation factor XI is associated with illness severity and coagulopathy in critically-ill septic patients

2019 Journal of Thrombosis and Thrombolysis
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Thromboembolic events in patients with severe inherited fibrinogen deficiency

2016 Journal of Thrombosis and Thrombolysis
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An allosteric disulfide bond is involved in enhanced activation of factor XI by protein disulfide isomerase.

2016 Journal of Thrombosis and Haemostasis
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Abnormal cytoplasmic extensions associated with active αIIbβ3 are probably the cause for macrothrombocytopenia in Glanzmann thrombasthenia-like syndrome.

2015 Blood Coagulation & Fibrinolysis
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The role of protein disulfide isomerase in the post-ligation phase of β3 integrin-dependent cell adhesion

2015 Thrombosis Research
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Persistent neonatal thrombocytopenia can be caused by IgA antiplatelet antibodies in breast milk of immune thrombocytopenic mothers

2015 Blood
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Natural and Artificial Mutations in αIIb Integrin Lead to a Structural Deformation of a Calcium-Binding Site

2014 Protein Journal
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Essential Thrombocythemia Patients with CALR Mutations Have Increased Platelet Activation Markers Compared to JAK2V617F Mutated Patients

2014 Blood
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Abnormal plasma clot structure and stability distinguish bleeding risk in patients with severe factor XI deficiency

2014 Journal of Thrombosis and Haemostasis
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Citations (63)