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E. A. Deitch

31

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412

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MESENTERIC LYMPH FROM BURNED RATS WITH EVIDENCE OF GUT INJURY IS CYTOTOXIC FOR ENDOTHELIAL CELLS.: 84

2002 Shock
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Sepsis and the septic response: principles of management

2002
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Shock Lymph Alters Endothelial Cell Biology and Endothelial Cell-Leukocyte Interaction: 23.

1998 Journal of Burn Care & Rehabilitation
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Interruption of Mesenteric Lymph Reduces Lung Injury Following Hemorrhagic Shock: 115.

1998 Journal of Burn Care & Rehabilitation
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A Thermally Induced But Not a Chemically Induced Heat Shock Response Protects Against Bacterial Translocation and Villus Injury After LPS Challenge: 30.

1998 Journal of Burn Care & Rehabilitation
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INHIBITION OF NO BY L-NMMA REDUCES ENDOTOXIN-INDUCED INCREASES IN INTESTINAL PERMEABILITY TO BACTERIAL, iNOS ACTIVITY AND IL-6 PRODUCTION IN USSING CHAMBER MODEL.: 204

1998 Shock
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EFFECT OF ACUTE PHASE AND HEAT SHOCK STRESS ON APOPTOSIS IN INTESTINAL EPITHELIAL CELLS (CACO-2): 228

1996 Shock
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MODULATION OF GUT MICROFLORA PRODUCES LYMPH CYTOTOXIC TO ENDOTHELIAL CELLS.: 166

1996 Shock
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ENDOTOXIN INCREASES NITRIC OXIDE PRODUCTION WITH MESENTERIC LYMPH NODE, PLASMA AND INTESTINAL MUCUS IN RATS.: 196

1996 Shock
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ALTERATIONS IN THE INTESTINAL BACTERIAL FLORA MODULATE SYSTEMIC CYTOKINE RESPONSE TO HEMORRHAGIC SHOCK.: 6

1995 Shock
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