46. Alcoholic cardiomyopathy: A histochemical and electron microscopic study
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Alcoholic cardiomyopathy
It is not unusual for cardiomyopathies to present with conduction abnormalities. However, reports of the occurrence of complete heart block in alcoholic cardiomyopathy are infrequent. Brigden and Robinson1reported 50 cases of alcoholic cardiomyopathy; 19 had some form of conduction disease. Eight patients had left bundle-branch block (LBBB), four had right bundle-branch block (RBBB), and seven had a prolonged RP interval, and one patient showed complete heart block. Tobin et al2reviewed 30 cases of alcoholic cardiomyopathy. One patient had a prolonged PR interval, one had LBBB, and two patients were noted to have RBBB. Evans3noted a bundle-branch block pattern in two of 20 patients having alcoholic cardiomyopathy. It appears that the development of complete heart block in alcoholic cardiomyopathy is unusual. We report, herein, a patient who developed cardiomyopathy secondary to excessive beer consumption that was then complicated by complete heart block. Bundle of His
Alcoholic cardiomyopathy
Bundle branch block
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Alcoholic cardiomyopathy
Chronic alcoholic
Chronic alcoholism
Medical record
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Alcoholic cardiomyopathy
Dilated Cardiomyopathy
Excessive alcohol consumption
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Background: Cardiomyopathy is a heart muscle disorder so which the heart cannot contract optimally. Alcoholic cardiomyopathy is a type of cardiomyopathy that dilates due to long-term alcohol consumption. The prevalence of alcoholic cardiomyopathy is higher in men than women. The age group with the most alcoholic cardiomyopathy was 45 to 74 year. Death from alcoholic cardiomyopathy had an autopsy result marked muscle dilatation. A toxicological examination can show the level of alcohol consumed. The cause of death in alcoholic cardiomyopathy is muscle dilatation due to chronic ethanol abuse (Dolinak, 2005). Islam expressly forbids something that makes losing mind, like alcohol. Alcohol has many disadvantages for the consumer in the form of loss of mind and turning away from Allah. Objective: This report aims to learn more about the definition, pathophysiology, clinical manifestation, and treatment of Alcoholic Cardiomyopathy. Also, Knowing and explaining Alcoholic Cardiomyopathy in terms of death and Islamic views. Methods: The design of this research is a literature review or library review, which is a systematic, direct, and reproducible research method by identifying, evaluating, and synthesizing published research. Result: Alcoholic cardiomyopathy is a clinical diagnosis made in a patient with a set of findings that include a history of excessive alcohol consumption, possible physical signs of alcohol abuse (e.g., parotid disease, telangiectasia or spider angiomata, altered mental status, cirrhosis), heart failure, and other evidence consistent with dilated cardiomyopathy. People who frequently consume alcohol can cause nutritional disorders, especially vitamin B1 deficiency can cause cardiomyopathy. Alcohol will also cause abnormalities in the structure and function of mitochondria in heart cells. These abnormalities include enlarged mitochondrial structure, decreased metabolism, lack of several enzymes decreased ion transport, increased calcium flow, glycogen accumulation, and decreased ATP production. Conclusion: Cardiomyopathy is a heart muscle function disorder characterized by the loss of the ability of the heart muscle to pump blood so that the blood supply is not optimal and results in death. The most common cause is chronic alcohol consumption, which causes dilatation of the heart muscle. At the same time, alcohol is an ingredient that is forbidden to be consumed by Muslims because of its more significant impact.
Alcoholic cardiomyopathy
Dilated Cardiomyopathy
Alcohol abuse
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Physical, mental, and emotional stressors have been well known to adversely affect cardiac function. A rare complication of such stressors is stress cardiomyopathy, otherwise known as takotsubo cardiomyopathy. First identified in Japan in the 1990s, takotsubo cardiomyopathy classically presents with systolic dysfunction and apical ballooning. In this report, we present the case of a patient with a medical history of alcohol abuse who presented to the emergency department after being found unresponsive by her family. Transthoracic echocardiography revealed takotsubo cardiomyopathy, which was likely secondary to alcohol withdrawal. Alcohol withdrawal causes an imbalance between various neurotransmitters such as GABA and glutamate. This imbalance caused autonomic overactivity, which manifested as stress cardiomyopathy.
Alcoholic cardiomyopathy
Stressor
Alcohol abuse
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Alcoholic cardiomyopathy
Dilated Cardiomyopathy
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Alcoholic cardiomyopathy
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Left ventricular non-compaction cardiomyopathy, often known as LVNC, is a form of congenital cardiomyopathy that is extremely uncommon. It is a condition that may be identified by an elevated number of endomyocardial trabeculations as well as an increase in their prominence. Alcoholic cardiomyopathy, also known as ACM, is a non-ischemic form of dilated cardiomyopathy that is characterized by contractile failure and an enlargement of the heart ventricles. It is not entirely known whether or not there is a clinically significant overlap in phenotypic characteristics between the two illnesses. We report a patient who had previously been diagnosed with ACM and who had cardiac MRI results that fit the criteria for both LVNC and ACM.
Alcoholic cardiomyopathy
Left ventricular noncompaction
Dilated Cardiomyopathy
Restrictive cardiomyopathy
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Alcoholic cardiomyopathy
Pathogenesis
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The purpose of the work was to analyze the current literature data on the morphology and thanatogenesis of alcoholic cardiomyopathy, as well as to determine further prospects for research on this issue. Domestic and foreign literary sources devoted to sudden death, pathomorphology of alcoholic cardiomyopathy, immunohistochemical and biochemical methods of its diagnosis were studied. As a result of the analysis, the essence of thanatogenesis in alcoholic cardiomyopathy is revealed, the prospect of immunohistochemical and morphometric methods is outlined.
According to the literature data, based on the results of the studies, an approximate scheme of thanatogenesis and subsequent scientific directions for the study of investigative deaths in alcoholic cardiomyopathy, in search of other diseases, can be built. In the Australian literature, the benefits of immunohistochemical are used to elucidate the antecedent pathochemical consequences. Also, in relation to the diagnosis of death from alcoholic cardiomyopathy, thanatogenetic analysis can be provided, since the sequence of development of fatally significant events in the form of various sudden deaths is not the same. In the interpretation of the processes occurring at the end of thanatogenesis in the myocardium, polarization microscopic examination is indispensable. I would like to note a number of biochemical changes in alcoholic cardiomyopathy, which demonstrate some features of the course of metabolic processes in this pathology, especially since this method and the immunohistochemical study are more in line with the modern requirements of evidence-based medicine.
As a result of the literature analysis, the essence of thanatogenesis in alcoholic cardiomyopathy was revealed, the prospect of immunohistochemical and morphometric methods was outlined.
Alcoholic cardiomyopathy
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