A resting EEG study of neocortical hyperexcitability and altered functional connectivity in fragile X syndrome
2017
Background
Cortical hyperexcitability due to abnormal fast-spiking inhibitory interneuron function has been documented in fmr1 KO mice, a mouse model of the fragile X syndrome which is the most common single gene cause of autism and intellectual disability.
Keywords:
- Correction
- Source
- Cite
- Save
- Machine Reading By IdeaReader
62
References
70
Citations
NaN
KQI