GW28-e0427 MiR-155 in macrophage regulates cardiac function and fibrosis after myocardial infarction possibly through SOCS1-dependent inflammatory response

Ventricular remodeling after myocardial infarction (MI) is the prominent cause of heart failure. Previous studies confirmed that the macrophage activation post-MI affects the remodeling of ventricle. We hypothesized that there is a certain miRNA regulating inflammatory factor that is involved in the