THE ETIOLOGY OF NONPARALYTIC OCULAR IMBALANCE: SOME ORIGINAL CONCEPTIONS AND INTERPRETATIONS BASED ON THE PHYSIOLOGY AND PSYCHOLOGY OF OCULAR MOVEMENTS

In the presession volume of last year's meeting, in a paper published in the March issue of Knapp's Archives , 1 I laid the foundation for a new consideration of this still clouded problem by a critique on paralytic types of ocular muscle imbalance. That was a serious effort to get a new point of attack on the problem, and this discussion is a follow-up one. It is the hope that the value of considering the question from the standpoint of levels of control will be hereinafter apparent. In the above-mentioned critique on paralytic types and by virtue of a close study of clinical pathology, certain fundamental points were demonstrated, and evidences in the recent literature were cited in proof of levels of control of the ocular movements. 2 These so-called levels of control are in the light of present knowlege quite accurately oriented for the high or third, and first