Age-related injury responses of human oligodendrocytes to metabolic insults: link to BCL-2 and autophagy pathways.

Myelin destruction and oligodendrocyte (OL) death consequent to metabolic stress is a feature of CNS disorders across the age spectrum. Using cells derived from surgically resected tissue, we demonstrate that young (

Keywords:

• Cancer research
• Multiple sclerosis
• Oligodendrocyte
• Biology
• Programmed cell death
• Cell
• Myelin
• Autophagy
• Progenitor cell
• Apoptosis

• Correction
• Source
• Cite
• Save
• Machine Reading By IdeaReader