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Loss-of-function mutations in SLC30A8 protect against type 2 diabetes
Loss-of-function mutations in SLC30A8 protect against type 2 diabetes
2014
Jason Flannick
Gudmar Thorleifsson
Nicola L. Beer
Suzanne B.R. Jacobs
Niels Grarup
Noël P. Burtt
Anubha Mahajan
Christian Fuchsberger
Gil Atzmon
Rafn Benediktsson
John Blangero
Donald W. Bowden
Ivan Brandslund
Julia Brosnan
Frank Burslem
John C. Chambers
Yoon Shin Cho
Cramer Christensen
Desirée A. Douglas
Ravindranath Duggirala
Zachary Dymek
Yossi Farjoun
Timothy Fennell
Pierre Fontanillas
Tom Forsén
Stacey Gabriel
Benjamin Glaser
Daniel F. Gudbjartsson
Craig L. Hanis
Torben Hansen
Ástradur B. Hreiðarsson
Kristian Hveem
Erik Ingelsson
Bo Isomaa
Stefan Johansson
Torben Jørgensen
Marit E. Jørgensen
Sekar Kathiresan
Augustine Kong
Jaspal S. Kooner
Jasmina Kravic
Markku Laakso
Jong-Young Lee
Lars Lind
Cecilia M. Lindgren
Allan Linneberg
Gisli Masson
Thomas Meitinger
Karen L. Mohlke
Anders Molven
Andrew P. Morris
Shobha Potluri
Rainer Rauramaa
Rasmus Ribel-Madsen
Ann Marie Richard
Tim Rolph
Veikko Salomaa
Ayellet V. Segrè
Hanna Skaerstrand
Valgerdur Steinthorsdottir
Heather M. Stringham
Patrick Sulem
E. Shyong Tai
Yik Ying Teo
Tanya M. Teslovich
Unnur Thorsteinsdottir
Jeff K. Trimmer
Tiinamaija Tuomi
Jaakko Tuomilehto
Fariba Vaziri-Sani
Benjamin F. Voight
James G. Wilson
Michael Boehnke
Mark I. McCarthy
Pål R. Njølstad
Oluf Pedersen
Leif Groop
David R. Cox
Kari Stefansson
David Altshuler
Keywords:
Internal medicine
Endocrinology
Loss function
Type 2 diabetes
SLC30A8
Medicine
Correction
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