A distinct mechanism of senescence activation in amnion epithelial cells by infection, inflammation, and oxidative stress
2018
Problem
We investigated p38MAPK activation-induced fetal membrane cell senescence in response to inflammation (tumor necrosis factor-alpha [TNF-α]) and infection (lipopolysaccharide [LPS]), factors associated with spontaneous preterm birth.
Keywords:
- Correction
- Source
- Cite
- Save
- Machine Reading By IdeaReader
46
References
40
Citations
NaN
KQI