Absent SEP during therapeutic hypothermia did not reappear after re-warming in comatose patients following cardiac arrest.

P arrest brain injury is a common cause of morbidity and mortality.1-3 Thus, the early prediction of neurological outcome after cardiopulmonary resuscitation (CPR) focuses on a poor outcome, which is no better than a vegetative state. In 2006, the American Academy of Neurology (AAN) 4 defined clinical and instrumental reliable predictors of a poor neurological outcome in coma after cardiac arrest (CA). Shortly before the publication of these practice parameters, two randomised controlled trials showed that induced therapeutic hypothermia (32-34 °C) (TH) decreases mortality and improves neurologic outcome in adult comatose survivors of outof-hospital cardiac arrest.5, 6 Soon thereafter, this treatment was incorporated in the guidelines of Absent SEP during therapeutic hypothermia did not reappear after re-warming in comatose patients following cardiac arrest