Evolution of dopants and defects in silicon under various annealing sequences

Several decades of research into understanding the mechanisms responsible for diffusion and activation of group-III acceptor and group-V donor impurities in silicon has been driven by the technological need for creating electrical junctions in the S/D and extensions of transistors in semiconductor integrated circuits. It is now conclusively known that anomalous diffusion of implanted impurities during annealing results from their interaction with the non-conservative evolution of excess point-defect damage created by the original implantation, and the effect annealing ambients, co-impurities, interfaces and surfaces have on that evolution [1–8]. Early experiments shown in Ref. [2] conclusively proved this by etching away the surface region containing implant damage before annealing, and demonstrating that the anomalous behavior disappeared. Starting from the as-implanted damage, the nucleation, growth and dissolution of a sequence of larger defects at the expense of smaller ones during the anneal, sets the point defect flux and super-saturation levels (the ratio of point defect concentration to its temperature equilibrium value), which then determine the magnitude and durations of the various phases of anomalous diffusion [9].