Outcome of blood pressure and renal function in patients with renal artery stenosis after stenting.

2007 
Renal artery stenosis (RAS) is a major cause of renovascular hypertension. RAS is also increasingly being recognized as the cause of chronic renal insufficiency and end-stage renal disease. In the general hypertensive population, its prevalence varies between 1% and 5%; however, this figure rises to between 10% and 45% in patients with acute, severe or refractory hypertension (1–3). The most common causes of RAS are atherosclerosis (90%) and fibromuscular dysplasia (8% to 9%) (2). Atherosclerotic RAS tends to occur in older individuals with risk factors for vascular disease, particularly diabetes mellitus (DM). RAS can be treated by conventional balloon angioplasty, angioplasty with stenting or bypass surgery (2,4,5). The aim of the present study was to examine the response of systolic and diastolic blood pressure (BP) and renal function after renal artery stenting at three months, six months, one year and last follow-up.
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