The inhibition of 2-arachidonoyl-glycerol (2-AG) biosynthesis, rather than enhancing striatal damage, protects striatal neurons from malonate-induced death: a potential role of cyclooxygenase-2-dependent metabolism of 2-AG

Sara Valdeolivas,María Ruth Pazos,T. Bisogno,Francesco Piscitelli,Fabio Arturo Iannotti,M. Allarà,Onintza Sagredo,V. Di Marzo,J.J. Fernández-Ruiz

The inhibition of 2-arachidonoyl-glycerol (2-AG) biosynthesis, rather than enhancing striatal damage, protects striatal neurons from malonate-induced death: a potential role of cyclooxygenase-2-dependent metabolism of 2-AG

2013

Sara Valdeolivas
María Ruth Pazos
T. Bisogno
Francesco Piscitelli
Fabio Arturo Iannotti
M. Allarà
Onintza Sagredo
V. Di Marzo
J.J. Fernández-Ruiz

The inhibition of 2-arachidonoyl-glycerol (2-AG) biosynthesis, rather than enhancing striatal damage, protects striatal neurons from malonate-induced death: a potential role of cyclooxygenase-2-dependent metabolism of 2-AG

Keywords:

Cell biology
Brain-derived neurotrophic factor
gamma-Aminobutyric acid
Endocannabinoid system
Programmed cell death
Cyclooxygenase
Cytoprotection
Malonate
Biology
Monoacylglycerol lipase
Neuroprotection
Striatum
Biochemistry
Cannabinoid receptor type 2
Pharmacology
JZL184
Neurotoxicity

Correction
Source
Cite
Save
Machine Reading By IdeaReader

References

Citations