Presentación precoz de crisis y evolución de la epilepsia en la hemorragia intraparenquimatosa cerebral

hemorrhage, 18 (6.3%), with no previous epilepsy, had crises whilst being followed-up for a period of between 2 and 7 years. In 14 cases the hematoma was lobar and 4 involved the basal ganglia or thalamus. In 8 cases (2.8% of all hemorrhage), these crises occurred during the first 24 hours, or as a first symptom of intraparenchymatous hemorrhage. One patient presented with status epilepticus with generalized crises and two had subentrant secondarily generalized partial crises at the time of the ict us. Treatment with anti-epileptic drugs was started in 13 patients. Twelve patients (4.2% of the hemorrhages) developed symptomatic epilepsy with partial crises with or without secondary generalization. Results and conclusions. The maximum rate of recurrence was four crises per year. However, in one patient, reduction of the dose of medication led to the appearance of stat us epilepticus. Patients with crises of late onset developed epilepsy more often than those who had early crises. In those with cr ises