Г.Р. ХАСАНОВА, О.И. БИККИНИНА, В.А. АНОХИН, C.В. ХАЛИУЛЛИНА, М.Ю. ЯКОВЛЕВ Êàçàíñêèé ãîñóäàðñòâåííûé ìåäèöèíñêèé óíèâåðñèòåò Ðåñïóáëèêàíñêèé öåíòð ïî ïðîôèëàêòèêå è áîðüáå ñî ÑÏÈÄ è èíôåêöèîííûìè çàáîëåâàíèÿìè ÌÇ ÐÒ, ã. Êàçàíü Ðîññèéñêèé ãîñóäàðñòâåííûé ìåäèöèíñêèé óíèâåðñèòåò, ã. Ìîñêâà Êèøå÷íûé ýíäîòîêñèí êàê âåðîÿòíûé èíäóêòîð ñèñòåìíîãî âîñïàëèòåëüíîãî îòâåòà ïðè

Endotoxinemia and antiendotoxin humoral immune response in 138 patients with HIV infection was evaluated. Elevated levels of endotoxin are set and the deviations from “normal” concentrations of antibodies to glycolipids and lipopolysaccharide E. Coli O14 in the blood of most HIV-infected patients. Along with the indicators of the “chronic endotoxic aggression,” a subset of patients identifi ed laboratory markers of systemic infl ammatory response syndrome (proinfl ammatory interleukins 1