Hemorrhagic shock alters fracture callus composition and activates the IL6 and RANKL/OPG pathway in mice

BackgroundFracture and hemorrhagic shock often lead to impaired fracture healing. To elucidate underlying pathogenesis, this study aimed to analyze histological properties during fracture healing after hemorrhagic shock and involved signaling pathways in mice.METHODSMale C57BL/6NCrl mice were assign