Neutrophil-derived advanced glycation end products-Nε-(carboxymethyl) lysine promotes RIP3-mediated myocardial necroptosis via RAGE and exacerbates myocardial ischemia/reperfusion injury.

Ji’e Yang,Feng Zhang,Huairui Shi,Yang Gao,Zhen Dong,Leilei Ma,Xiaolei Sun,Xiao Li,Suchi Chang,Zeng Wang,Ya’nan Qu,Hua Li,Kai Hu,Aijun Sun,Junbo Ge

Neutrophil-derived advanced glycation end products-Nε-(carboxymethyl) lysine promotes RIP3-mediated myocardial necroptosis via RAGE and exacerbates myocardial ischemia/reperfusion injury.

2019

Ji’e Yang
Feng Zhang
Huairui Shi
Yang Gao
Zhen Dong
Leilei Ma
Xiaolei Sun
Xiao Li
Suchi Chang
Zeng Wang
Ya’nan Qu
Hua Li
Kai Hu
Aijun Sun
Junbo Ge

Ne-(carboxymethyl) lysine (CML), the major member of advanced glycation end products, was widely studied in diabetic complications and aging-associated diseases. However, the impact of CML on myoca...

Keywords:

Programmed cell death
Lysine
Reperfusion injury
Necroptosis
Diabetes mellitus
Internal medicine
Myocardial infarction
Biology
Endocrinology
Ischemia
Glycation
myocardial ischemia

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