The allosteric potentiation of nicotinic acetylcholine receptors by galantamine ameliorates the cognitive dysfunction in beta amyloid25-35 i.c.v.-injected mice: involvement of dopaminergic systems.

Dayong Wang,Yukihiro Noda,Yuan Zhou,Akihiro Mouri,Hiroyuki Mizoguchi,Atsumi Nitta,Weiduo Chen,Toshitaka Nabeshima

The allosteric potentiation of nicotinic acetylcholine receptors by galantamine ameliorates the cognitive dysfunction in beta amyloid25-35 i.c.v.-injected mice: involvement of dopaminergic systems.

2007

Dayong Wang
Yukihiro Noda
Yuan Zhou
Akihiro Mouri
Hiroyuki Mizoguchi
Atsumi Nitta
Weiduo Chen
Toshitaka Nabeshima

The Allosteric Potentiation of Nicotinic Acetylcholine Receptors by Galantamine Ameliorates the Cognitive Dysfunction in Beta Amyloid 25–35 I.c.v.-Injected Mice: Involvement of Dopaminergic Systems

Keywords:

Psychology
Acetylcholine receptor
Nicotinic agonist
Nicotinic acetylcholine receptor
Neurotransmitter
Long-term potentiation
Endocrinology
Internal medicine
Galantamine
Pharmacology
Dopaminergic
Acetylcholinesterase
Cholinergic
Neuroscience
Mecamylamine
Muscarinic acetylcholine receptor
Receptor antagonist

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