Acetylation preserves retinal ganglion cell structure and function in a chronic model of ocular hypertension.

Oday Alsarraf,Jie Fan,Mohammad Dahrouj,C. James Chou,Phillip W. Yates,Craig E. Crosson

Acetylation preserves retinal ganglion cell structure and function in a chronic model of ocular hypertension.

2014

Oday Alsarraf
Jie Fan
Mohammad Dahrouj
C. James Chou
Phillip W. Yates
Craig E. Crosson

Purpose. The current studies investigate if the histone deacetylase (HDAC) inhibitor, valproic acid (VPA), can limit retinal ganglion cell (RGC) degeneration in an ocular-hypertensive rat model.

Keywords:

Intraocular pressure
Acetylation
Retinal ganglion cell
Retina
Ocular hypertension
Electroretinography
Histone deacetylase
Anatomy
Valproic Acid
Biology
Retinal
hypertonic saline
Histone H3 acetylation

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